Key Cellular Mechanism Driving Alzheimer’s Disease
The Neuroscience Initiative is engaged in interdisciplinary programs with other initiatives and other institutions to implement interdisciplinary approaches for the study of environmental influences on brain function and behavior and to develop transformative technologies and advanced platforms aimed at promoting mental health.
- January 17, 2025
ASRC Researchers Tackle Alzheimer’s and Plastic Pollution with Cutting-Edge Science - December 23, 2024
New Research Identifies Key Cellular Mechanism Driving Alzheimer’s Disease - November 4, 2024
New Research Identifies Ways to Protect Neurons From the Negative Effect of High-Fat Diet on Multiple Sclerosis Progression - September 3, 2024
New Study Uncovers Key Mechanisms Responsible for the Transformation of Adult Progenitors Into Brain Tumors - August 12, 2024
Adult Oligodendrocyte Progenitor Cell Study Opens Doors for Advanced Myelin Repair Therapies
- April 3, 2025 @ 12:00 pm
Neuroscience Spring 2025 Seminar Series: “Unraveling the Behavioral Complexity of Social Dominance Hierarchies in Mice.” - May 1, 2025 @ 12:00 pm
Neuroscience Spring 2025 Seminar Series: “Early-life gut inflammation drives sex-dependent shifts in the microbiome-endocrine-brain axis.” - June 5, 2025 @ 12:00 pm
Neuroscience Spring 2025 Seminar Series: “The neural circuits underlying overgeneralization.”
M. Pruvost, H.-J. Park, C. Habermacher, M. Li, M. C. Angulo, J. Liu, P. Casaccia. The Histone Methyltransferases EHMT1 and EHMT2 Repress the Expression of Genes Related to Excitability and Cell Death in Oligodendrocyte Progenitors. Glia, 2025. DOI: https://doi.org/10.1002/glia.70014.
A. Flury, L. Aljayousi, H.-J. Park, M. Khakpour, J. Mechler, S. Aziz, J. D. McGrath, P. Deme, C. Sandberg, F. González Ibáñez, O. Braniff, T. Ngo, S. Smith, M. Velez, D. M. Ramirez, D. Avnon-Klein, J. W. Murray, J. Liu, M. Parent, S. Mingote, et al. A Neurodegenerative Cellular Stress Response Linked to Dark Microglia and Toxic Lipid Secretion. Neuron, 2025, 113 (4), 554-571.e14. DOI: https://doi.org/10.1016/j.neuron.2024.11.018.
M. Amatruda, D. Marechal, M. Gacias, M. Wentling, S. Turpin‐Nolan, J. Morstein, M. Moniruzzaman, J. C. Brüning, N. J. Haughey, D. H. Trauner, P. Casaccia. Neuroprotective Effect of Neuron‐specific Deletion of the C16 Ceramide Synthetic Enzymes in an Animal Model of Multiple Sclerosis. Glia, 2024, glia.24631. DOI: https://doi.org/10.1002/glia.24631.