Key Cellular Mechanism Driving Alzheimer’s Disease
The Neuroscience Initiative is engaged in interdisciplinary programs with other initiatives and other institutions to implement interdisciplinary approaches for the study of environmental influences on brain function and behavior and to develop transformative technologies and advanced platforms aimed at promoting mental health.
- January 17, 2025
ASRC Researchers Tackle Alzheimer’s and Plastic Pollution with Cutting-Edge Science - December 23, 2024
New Research Identifies Key Cellular Mechanism Driving Alzheimer’s Disease - November 4, 2024
New Research Identifies Ways to Protect Neurons From the Negative Effect of High-Fat Diet on Multiple Sclerosis Progression - September 3, 2024
New Study Uncovers Key Mechanisms Responsible for the Transformation of Adult Progenitors Into Brain Tumors - August 12, 2024
Adult Oligodendrocyte Progenitor Cell Study Opens Doors for Advanced Myelin Repair Therapies
M. Amatruda, D. Marechal, M. Gacias, M. Wentling, S. Turpin‐Nolan, J. Morstein, M. Moniruzzaman, J. C. Brüning, N. J. Haughey, D. H. Trauner, P. Casaccia. Neuroprotective Effect of Neuron‐specific Deletion of the C16 Ceramide Synthetic Enzymes in an Animal Model of Multiple Sclerosis. Glia, 2024, glia.24631. DOI: https://doi.org/10.1002/glia.24631.
S. B. Siems, V. Gargareta, L. C. Schadt, V. Daguano Gastaldi, R. B. Jung, L. Piepkorn, P. Casaccia, T. Sun, O. Jahn, H. B. Werner. Developmental Maturation and Regional Heterogeneity but No Sexual Dimorphism of the Murine CNS Myelin Proteome. Glia, 2024, glia.24614. DOI: https://doi.org/10.1002/glia.24614.
D. Huang, A. Mela, N. V. Bhanu, B. A. Garcia, P. Canoll, P. Casaccia. PDGF-BB Overexpression in P53 Null Oligodendrocyte Progenitors Increases H3K27me3 and Induces Transcriptional Changes Which Favor Proliferation. Neoplasia, 2024, 57, 101042. DOI: https://doi.org/10.1016/j.neo.2024.101042.